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Moreover, in response to stress, p66Shc (which functions in the intracellular pathways that convert oxidative signals into apoptosis) translocates to mitochondria to directly stimulate hydrogen peroxide generation by transferring electrons to cytochrome c

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In necroptosis, three cysteine residues (C257, C268 and C586) in RIPK1 form intermolecular disulfide bonds, which ultimately induce autophosphorylation of serine residue 161 (S161) of RIPK1