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Pro-inflammatory cytokines, such as TNF-α, IL-1β and IL-6, and MMPs secreted from SFs result in the further activation of immune cells, and the inflammatory cycle created by them leads to the pathological characters and systemic manifestations of patients with RA.[21] Thus, targeting these cytokines as a therapeutic intervention has been regarded as a rational and efficient choice, and examples of such therapeutics include sarilumab, tocilizumab, infliximab, and adalimumab.[22] The MMPs produced from SFs usually contribute to the destruction of cartilage seen in RA.[23] The present RT-qPCR and ELISA results provided evidence that the levels of TNF-α and IL-6 were significantly decreased by GSH
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During a Herxheimer reaction, toxins will be converted to compounds that can dissolve in water
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